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Inflammation has been defined as the local reaction of the body to irritation, its object being the removal of the irritant by neutralization, destruction, or encapsulation. This is attempted by its characteristic feature, exudation, which is defined as the outpouring of adventitious substances of a fluid or cellular nature. The secondary features of the inflammatory process are degeneration and proliferation. The degeneration, or disintegration of the tissues, is dependent upon the virulence of the irritant and the resistance of the tissues. Proliferation, a manifestation of repair, is dependent upon the vitality of the tissues. Inflammation is not synonymous with infection, the latter being defined as the invasion of the tissues by pathogenic organisms in such a way that injury followed by reactive phenomena results. Non-infectious substances such as red blood cells, toxins, necrotie tissue and tumors often provoke an inflammatory response. The abnormal presence of "inflammatory cells," such as polymorphonuclear leucocytes and round cells, is generally looked upon as the cardinal feature of the inflammatory process. The inflammatory conditions of the nervous system have been variously classified according to: (a) the site of the inflammatory process: (b) the outstanding morbid anatomic feature; (e) the pathogenic or inciting agent or the nature of the systemic disorder; (d) the outstanding clinical symptom: (e) the time required for the evolution of the inflammatory process; and (f) the names of persons or places prominent in the history of the disorder. The author focuses on morbid anatomic types of meningo-encephalomyelitis, etiological types of meningo-encephalomyelitis, and blastomatous states resembling meningo-encephalomyelitis. (PsycInfo Database Record (c) 2022 APA, all rights reserved)
Published: Nov 13, 2006
Keywords: inflammation; neuropathology; nervous system inflammation; meningo-encephalomyelitis; etiology
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