Get 20M+ Full-Text Papers For Less Than $1.50/day. Start a 14-Day Trial for You or Your Team.

Learn More →

Hepatocellular transplantation for metabolic deficiencies: decrease of plasms bilirubin in Gunn rats.

Hepatocellular transplantation for metabolic deficiencies: decrease of plasms bilirubin in Gunn... A sustained decrease of plasma bilirubin concentrations occurred in homozygous recessive Gunn rats lacking the enzyme uridine diphosphate glucuronyltransferase following infusion into the portal vein of hepatocytes from heterozygous nonjaundiced Gunn rats possessing the enzyme. Transplantation of cells capable of continuous enzyme production could be an effective mode of therapy for congenital enzyme deficiency diseases. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Science (New York, N.Y.) Pubmed

Hepatocellular transplantation for metabolic deficiencies: decrease of plasms bilirubin in Gunn rats.

Science (New York, N.Y.) , Volume 192 (4242): -887 – Aug 2, 1976

Hepatocellular transplantation for metabolic deficiencies: decrease of plasms bilirubin in Gunn rats.


Abstract

A sustained decrease of plasma bilirubin concentrations occurred in homozygous recessive Gunn rats lacking the enzyme uridine diphosphate glucuronyltransferase following infusion into the portal vein of hepatocytes from heterozygous nonjaundiced Gunn rats possessing the enzyme. Transplantation of cells capable of continuous enzyme production could be an effective mode of therapy for congenital enzyme deficiency diseases.

Loading next page...
 
/lp/pubmed/hepatocellular-transplantation-for-metabolic-deficiencies-decrease-of-ValMSTxRCm

References

References for this paper are not available at this time. We will be adding them shortly, thank you for your patience.

ISSN
0036-8075
DOI
10.1126/science.818706
pmid
818706

Abstract

A sustained decrease of plasma bilirubin concentrations occurred in homozygous recessive Gunn rats lacking the enzyme uridine diphosphate glucuronyltransferase following infusion into the portal vein of hepatocytes from heterozygous nonjaundiced Gunn rats possessing the enzyme. Transplantation of cells capable of continuous enzyme production could be an effective mode of therapy for congenital enzyme deficiency diseases.

Journal

Science (New York, N.Y.)Pubmed

Published: Aug 2, 1976

There are no references for this article.